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THE COMPARISON OF WEIGHT TRAINING AND ENDURANCE EXERCISE TOWARDS THE LEVEL DAMAGE OF MUSCLE CELLS ( fsTnI)
Fuad Noor Heza(a*), Ngadiman(a), Rifqi Festiawan(a), Ajeng Dian Purnamasari (a), Indra Jati Kusuma (a), Bayu Suko Wahono (a), Winarno (b).

a) Physical Education, Faculty of Health Science, Jenderal Soedirman University.
fuad.heza[at]unsoed.ac.id
b) Physical Education, Faculty of Sport Science, State University of Malang.


Abstract

Muscle damage is the failure of each level to maintain a balance between stress exposure and the response to stress. fsTnI found in serum is a special marker for muscle injury due to exercise or exercise induced muscle damage. This study examines the extent of muscle cell damage caused by stressors of strenuous physical exercise with endurance training on levels of fast skeletal muscle Troponin-I (fsTnI). The method used in this study was experimental laboratories which were divided into two groups of weight training and endurance exercise in experimental animals of male wistar rats. The results of this study indicate that the t-test showed a significant difference of p <0.05 in the two groups. The mean analysis of Troponin-I fast skeletal muscle (fsTnI) levels compared the two groups, weight training showed higher than endurance exercise, which means that heavy exercise showed more damage in Troponin-I fast skeletal muscle (fsTnI). Weight training can cause interference with sarcomere. The structure of the sarcomere becomes unstable, the fibril protein breaks down and the structure of the sarcomere is damaged resulting in impaired function of muscle contraction. Severe physical exercise causes disruption of the sarcomere in elements of the cytoskeletal involved in channeling ATP, damage to cell membranes, loss of homeostasis from calcium ions and loss of function to produce ATP in muscles. It was concluded that weight training increases the risk of fsTnI muscle cell damage more than endurance exercise.

Keywords: weight training, endurance, muscle cells, fsTnI.

Topic: Sport Science

Plain Format | Corresponding Author (Fuad Noor Heza)

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